INT186448

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Context Info
Confidence 0.02
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.23
Pain Relevance 0.92

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Prkcg, Trib3, Creb1) nucleolus (Trib3, Creb1) nucleoplasm (Creb1)
mitochondrion (Creb1) intracellular (Prkcg) DNA binding (Creb1)
Anatomy Link Frequency
spinal cord 2
Prkcg (Rattus norvegicus)
Trib3 (Rattus norvegicus)
Creb1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Kinase C 18 100.00 Very High Very High Very High
Spinal cord 15 98.76 Very High Very High Very High
long-term potentiation 5 96.52 Very High Very High Very High
central sensitization 13 94.48 High High
qutenza 29 80.64 Quite High
spinothalamic 3 69.08 Quite High
Pain 5 55.20 Quite High
c fibre 1 36.80 Quite Low
anesthesia 1 35.04 Quite Low
intrathecal 6 30.00 Quite Low
Disease Link Frequency Relevance Heat
Hypersensitivity 1 68.80 Quite High
Nociception 17 57.52 Quite High
Pain 2 55.20 Quite High
Injury 1 5.00 Very Low Very Low Very Low
INFLAMMATION 1 5.00 Very Low Very Low Very Low
Nervous System Injury 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In summary, the findings from our investigation provide strong evidence that MAP kinase, PKA and PKC participate in the regulation of CREB phosphorylation status in the spinal cord following peripheral noxious stimulation.
PKC Regulation (participate) of kinase Regulation (regulation) of CREB in spinal cord associated with kinase c and spinal cord
1) Confidence 0.02 Published 2005 Journal Mol Pain Section Body Doc Link PMC1224868 Disease Relevance 0.23 Pain Relevance 0.92

General Comments

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