INT187377

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Context Info
Confidence 0.09
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.89
Pain Relevance 0.51

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (TH1L) nucleus (TH1L)
TH1L (Homo sapiens)
Pain Link Frequency Relevance Heat
Cannabinoid 118 99.08 Very High Very High Very High
Cannabinoid receptor 8 94.72 High High
Multiple sclerosis 14 66.20 Quite High
cannabis 81 64.24 Quite High
Inflammation 8 37.84 Quite Low
Central nervous system 1 35.96 Quite Low
Endocannabinoid 32 5.00 Very Low Very Low Very Low
Delta-9-tetrahydrocannabinol 23 5.00 Very Low Very Low Very Low
Pain 15 5.00 Very Low Very Low Very Low
alcohol 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Infection 6 89.28 High High
Death 8 78.72 Quite High
Disease 23 69.28 Quite High
General Immunology 3 68.76 Quite High
Mycobacterial Infection 2 67.28 Quite High
Multiple Sclerosis 20 66.20 Quite High
Autoimmune Disease 2 61.24 Quite High
Spasticity 2 52.04 Quite High
INFLAMMATION 8 37.84 Quite Low
Neurodegenerative Disease 1 31.56 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
While often classified as immune inhibitors, cannabinoids actually promote the Th2 response while they inhibit the Th1 response.
Negative_regulation (inhibit) of Regulation (response) of Th1 associated with cannabinoid
1) Confidence 0.09 Published 2005 Journal Harm Reduct J Section Body Doc Link PMC1261530 Disease Relevance 0.89 Pain Relevance 0.51

General Comments

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