INT187796

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.09
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.85
Pain Relevance 0.48

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell proliferation (Tnf) signal transduction (Tnf) extracellular space (Tnf)
extracellular region (Tnf) plasma membrane (Tnf) intracellular (Tnf)
Tnf (Mus musculus)
Il1 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 28 97.28 Very High Very High Very High
cINOD 2 85.20 High High
cytokine 21 77.12 Quite High
Inflammatory mediators 3 76.32 Quite High
glial activation 4 50.00 Quite Low
Hippocampus 9 5.00 Very Low Very Low Very Low
imagery 5 5.00 Very Low Very Low Very Low
long-term potentiation 3 5.00 Very Low Very Low Very Low
Inflammatory marker 2 5.00 Very Low Very Low Very Low
chemokine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Necrosis 2 100.00 Very High Very High Very High
Cancer 2 100.00 Very High Very High Very High
Disease 24 99.76 Very High Very High Very High
INFLAMMATION 35 97.28 Very High Very High Very High
Amyloid Plaque 16 92.40 High High
Death 3 82.84 Quite High
Cognitive Disorder 3 77.84 Quite High
Alzheimer's Dementia 16 69.32 Quite High
Cerebral Amyloid Angiopathy 1 58.44 Quite High
Amyloidosis 1 55.20 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In addition to histopathological and biochemical data, several proinflammatory genes have been linked to an increased risk for AD, including interleukin1 (Il-1) [12], interleukin 6 (Il-6) [13] and tumor necrosis factor alpha (TNF?)
tumor necrosis factor alpha Binding (interleukin1) of Il-1 associated with necrosis, cancer and disease
1) Confidence 0.09 Published 2005 Journal J Neuroinflammation Section Body Doc Link PMC1274341 Disease Relevance 1.85 Pain Relevance 0.48

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox