INT188609

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Context Info
Confidence 0.02
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.51
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (ACACA) mitochondrion (ACACA) small molecule metabolic process (ACACA)
ligase activity (ACACA) cytoplasm (ACACA)
ACACA (Homo sapiens)
Pain Link Frequency Relevance Heat
Glutamate 6 100.00 Very High Very High Very High
nMDA receptor 3 12.56 Low Low
Kinase C 1 5.12 Low Low
Central nervous system 12 5.00 Very Low Very Low Very Low
depression 9 5.00 Very Low Very Low Very Low
anticonvulsant 3 5.00 Very Low Very Low Very Low
gABA 3 5.00 Very Low Very Low Very Low
cva 2 5.00 Very Low Very Low Very Low
Migraine 1 5.00 Very Low Very Low Very Low
Neuropathic pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Encephalopathy 24 99.84 Very High Very High Very High
Congenital Anomalies 4 95.92 Very High Very High Very High
Urinary Tract Infection 1 83.92 Quite High
Partial Seizures 1 79.56 Quite High
Syndrome 4 67.08 Quite High
Toxicity 9 50.00 Quite Low
Convulsion 7 46.64 Quite Low
Neurologic Manifestations 1 45.76 Quite Low
Hepatotoxicity 42 11.88 Low Low
Chronic Disease 1 9.48 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
As acetyl-CoA stores are depleted, the synthesis of NAGA – an important cofactor of CPS I – from acetyl-CoA and glutamate by NAGA synthetase is decreased.
Negative_regulation (decreased) of Gene_expression (synthesis) of acetyl-CoA associated with glutamate
1) Confidence 0.02 Published 2005 Journal Crit Care Section Body Doc Link PMC1297603 Disease Relevance 0.51 Pain Relevance 0.08

General Comments

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