INT189702

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Context Info
Confidence 0.06
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.55
Pain Relevance 0.10

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (CHKB, TERT) nucleoplasm (TERT) DNA binding (TERT)
RNA binding (TERT) chromosome (TERT) nucleolus (TERT)
CHKB (Homo sapiens)
TERT (Homo sapiens)
Pain Link Frequency Relevance Heat
Kinase C 94 68.32 Quite High
agonist 8 5.00 Very Low Very Low Very Low
Central nervous system 2 5.00 Very Low Very Low Very Low
cINOD 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cancer 32 99.40 Very High Very High Very High
Increased Venous Pressure Under Development 2 71.80 Quite High
Apoptosis 6 66.24 Quite High
Repression 8 52.84 Quite High
Aging 8 5.00 Very Low Very Low Very Low
Reprotox - General 1 4 5.00 Very Low Very Low Very Low
Targeted Disruption 4 5.00 Very Low Very Low Very Low
Hyperplasia 2 5.00 Very Low Very Low Very Low
Cervical Cancer 2 5.00 Very Low Very Low Very Low
Genetic Translocation 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The Receptor Ck-dependent down-regulation of hTERT mRNA expression observed in the present study may cooperate with the other repressive mechanisms operational in PBMCs in order to maintain appropriate low levels of telomerase in these cells.
Ck Regulation (regulation) of Gene_expression (expression) of hTERT mRNA
1) Confidence 0.06 Published 2006 Journal BMC Cell Biol Section Body Doc Link PMC1351175 Disease Relevance 0.46 Pain Relevance 0
Activated Receptor Ck was found to down-regulate hTERT mRNA expression by repressing the transcription of c-myc gene.
Ck Regulation (regulate) of Gene_expression (expression) of hTERT mRNA
2) Confidence 0.06 Published 2006 Journal BMC Cell Biol Section Abstract Doc Link PMC1351175 Disease Relevance 0.09 Pain Relevance 0.10

General Comments

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