INT190857

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Context Info
Confidence 0.00
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.85
Pain Relevance 0.21

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Ppp1r17)
Anatomy Link Frequency
neurons 2
Ppp1r17 (Mus musculus)
Pain Link Frequency Relevance Heat
Kinase C 82 100.00 Very High Very High Very High
long-term potentiation 8 50.28 Quite High
nMDA receptor 3 48.88 Quite Low
Central nervous system 7 5.00 Very Low Very Low Very Low
Neurotransmitter 6 5.00 Very Low Very Low Very Low
Neurobehavioral 6 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
agonist 2 5.00 Very Low Very Low Very Low
Glutamate 2 5.00 Very Low Very Low Very Low
Hippocampus 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Virus Diseases 94 99.84 Very High Very High Very High
Infection 20 89.48 High High
Borna Disease 5 50.00 Quite Low
Disease 5 5.00 Very Low Very Low Very Low
Cognitive Disorder 3 5.00 Very Low Very Low Very Low
INFLAMMATION 3 5.00 Very Low Very Low Very Low
Sprains And Strains 3 5.00 Very Low Very Low Very Low
Neurodegenerative Disease 2 5.00 Very Low Very Low Very Low
Acquired Immune Deficiency Syndrome Or Hiv Infection 2 5.00 Very Low Very Low Very Low
Viral Infection 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In contrast, the phosphorylation of myristoylated alanine-rich C kinase substrate (MARCKS), a major PKC substrate in neurons [31], was severely impaired in BDV-infected neurons following treatment with glycine (Figure 4D).
Negative_regulation (impaired) of Phosphorylation (phosphorylation) of substrate in neurons associated with virus diseases and kinase c
1) Confidence 0.00 Published 2006 Journal PLoS Pathogens Section Body Doc Link PMC1401496 Disease Relevance 0.85 Pain Relevance 0.21

General Comments

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