INT19132
From wiki-pain
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Sentences Mentioned In
Key: | Protein | Mutation | Event | Anatomy | Negation | Speculation | Pain term | Disease term |
Transfection of mutant cDNAs into cultured cells results in the synthesis of immunoreactive sterol 27-hydroxylase protein with greatly diminished enzyme activity. | |||||||||||||||
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Adachi and Fukuyo [28] confirmed that wild and cultured G. toxicus produce precursors of CTXs (known as gambiertoxins) [29]. | |||||||||||||||
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In 2001, the total synthesis of CTX3C was achieved by Hirama et al. [139], based on highly convergent and efficient strategy for the assembly of structural fragments. | |||||||||||||||
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D CTX levels. | |||||||||||||||
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L CTX values below the mean of nonmetastasised controls (NC-patients), 52% of these patients had ? | |||||||||||||||
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L CTX levels alone showed a significant elevation in 58% of the metastatic patients, which probably reflects an increased bone turnover (increased resorption + increased formation) as an important feature in the majority of bone metastatic patients. | |||||||||||||||
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L CTX is reflected in the CTX ratios, providing an index of bone collagen half-life [9]. | |||||||||||||||
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L CTX and the NC+ patients had 2.2-fold higher levels of ? | |||||||||||||||
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D CTX ratio showed the highest average increases in accordance with the ability of the ? | |||||||||||||||
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L CTX levels and the ? | |||||||||||||||
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L CTX levels, and a 2.2-fold elevation was seen in NC+ patients, indicating that the more severe condition in the HC+ patients is reflected by the ? | |||||||||||||||
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This assay enabled the detection of P-CTXs and C-CTXs in the very low range of 0.0785.00 ng/mL (i.e., 7.8 × 10? | |||||||||||||||
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This assay enabled the detection of P-CTXs and C-CTXs in the very low range of 0.0785.00 ng/mL (i.e., 7.8 × 10? | |||||||||||||||
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Cerebrotendinous xanthomatosis is an inherited autosomal recessive disease caused by a mutation in the gene for the sterol 27-hydroxylase enzyme, which determines the accumulation of plasmatic cholestanol in various tissues. | |||||||||||||||
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Levels of both CTX-II and CTX-I, as markers of cartilage and bone degradation respectively, decreased by approximately 50% in the treatment group compared with baseline, and CTX-II levels were restored to premenopausal levels. | |||||||||||||||
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Levels of both CTX-II and CTX-I, as markers of cartilage and bone degradation respectively, decreased by approximately 50% in the treatment group compared with baseline, and CTX-II levels were restored to premenopausal levels. | |||||||||||||||
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Such OA may be referred to as a systemic, metabolic form of OA [1], which is supported by our finding that bone resorption, indicated by CTX-I levels, was higher in women than in men. | |||||||||||||||
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L CTX form and the older ? | |||||||||||||||
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Among the CTX ratios, the ? | |||||||||||||||
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L CTX form and the ? | |||||||||||||||
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General Comments
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