INT191537

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Context Info
Confidence 0.02
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.36
Pain Relevance 0.24

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (NLRP3) signal transduction (NLRP3) peptidase activity (Klk1c2)
cytoplasm (NLRP3)
Klk1c2 (Rattus norvegicus)
NLRP3 (Homo sapiens)
Pain Link Frequency Relevance Heat
Enkephalin 1 76.00 Quite High
bradykinin 1 69.84 Quite High
agonist 3 54.72 Quite High
Inflammatory response 3 53.92 Quite High
antagonist 3 53.68 Quite High
Inflammation 33 52.72 Quite High
cytokine 12 51.52 Quite High
chemokine 2 42.88 Quite Low
Herpes zoster virus 1 30.88 Quite Low
Angina 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Natriuresis 1 70.96 Quite High
Increased Venous Pressure Under Development 85 64.80 Quite High
INFLAMMATION 39 53.56 Quite High
Adhesions 10 41.88 Quite Low
Stress 28 34.44 Quite Low
Herpes Zoster 1 30.64 Quite Low
Cytomegalovirus Infection 2 30.04 Quite Low
Chlamydia Infection 2 28.04 Quite Low
Infection 2 27.52 Quite Low
Diabetes Mellitus 14 23.12 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Chymase, Carboxypeptidase, Cathepsin G and Tonin can generate AII from AI independently of ACE, and AII can also originate directly from angiotensinogen by non-renin enzymes, such as tissue plasminogen activator (t-AP), Cathepsin G and Tonin.
Tonin Binding (generate) of AII
1) Confidence 0.02 Published 2006 Journal Cardiovasc Diabetol Section Body Doc Link PMC1434727 Disease Relevance 0.36 Pain Relevance 0.24

General Comments

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