INT191981

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Context Info
Confidence 0.28
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.06
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Ide) mitochondrion (Ide) extracellular space (Ide)
plasma membrane (Sit1) ATPase activity (Ide) nucleus (Ide)
Ide (Mus musculus)
Sit1 (Mus musculus)
Pain Link Frequency Relevance Heat
vagus nerve 1 48.92 Quite Low
peripheral neuropathy 1 5.00 Very Low Very Low Very Low
Parenteral administration 1 5.00 Very Low Very Low Very Low
abdominal pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 18 59.84 Quite High
Diarrhoea 2 5.00 Very Low Very Low Very Low
Hypoglycemia 2 5.00 Very Low Very Low Very Low
Constipation 2 5.00 Very Low Very Low Very Low
Fecal Incontinence 1 5.00 Very Low Very Low Very Low
Gastric Motility Disorder 1 5.00 Very Low Very Low Very Low
Metabolic Disorder 1 5.00 Very Low Very Low Very Low
Abdominal Pain 1 5.00 Very Low Very Low Very Low
Neuropathic Pain 1 5.00 Very Low Very Low Very Low
Peripheral Neuropathy 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These findings indicate that exogenously administered insulin is solely responsible for significant acceleration of SIT, but with the higher doses of insulin elevated endogenous insulin levels produced cannot be ruled out in acceleration of SIT.
insulin Positive_regulation (responsible) of Positive_regulation (acceleration) of SIT
1) Confidence 0.28 Published 2006 Journal BMC Pharmacol Section Body Doc Link PMC1444921 Disease Relevance 0.06 Pain Relevance 0

General Comments

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