INT19304

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Context Info
Confidence 0.15
First Reported 1991
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 6
Disease Relevance 1.00
Pain Relevance 0.95

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (OXT) extracellular space (OXT) extracellular region (OXT)
response to stress (OXT) cytoplasm (OXT)
Anatomy Link Frequency
plasma 2
hypothalamus 2
OXT (Homo sapiens)
Pain Link Frequency Relevance Heat
narcan 8 99.98 Very High Very High Very High
Endogenous opioid 1 93.44 High High
Neuropeptide 90 90.28 High High
Opioid 2 87.44 High High
antagonist 1 87.16 High High
agonist 20 80.12 Quite High
Rostral ventrolateral medulla 5 79.28 Quite High
parabrachial 5 76.52 Quite High
Intracerebroventricular 10 75.52 Quite High
Central nervous system 20 71.84 Quite High
Disease Link Frequency Relevance Heat
Pressure Volume 2 Under Development 10 99.80 Very High Very High Very High
Natriuresis 5 84.72 Quite High
Premature Birth 10 69.24 Quite High
Heart Rate Under Development 5 66.40 Quite High
Nicotine Addiction 5 58.92 Quite High
Pre-term Labor 10 53.32 Quite High
Depression 5 5.00 Very Low Very Low Very Low
Increased Venous Pressure Under Development 5 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 5 5.00 Very Low Very Low Very Low
Stress 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Naloxone increases the angiotensin II stimulated rise of arginine vasopressin and oxytocin secretion in man.
Positive_regulation (increases) of Localization (secretion) of oxytocin associated with narcan
1) Confidence 0.15 Published 1991 Journal Neuroendocrinology Section Title Doc Link 2041583 Disease Relevance 0 Pain Relevance 0.50
However, the results of pronounced pressor responses in the present as well as previous study [15] after the i.c.v. carbachol do not support the possibility that volume mechanisms may contribute to AVP and OT release by central cholinergic stimulation in utero.
Positive_regulation (contribute) of Localization (release) of OT
2) Confidence 0.10 Published 2008 Journal BMC Dev Biol Section Body Doc Link PMC2570685 Disease Relevance 0.16 Pain Relevance 0.03
A variety of signals, including the central cholinergic stimulation, can cause release of AVP and OT in adults [13,14].
Positive_regulation (cause) of Localization (release) of OT
3) Confidence 0.09 Published 2008 Journal BMC Dev Biol Section Body Doc Link PMC2570685 Disease Relevance 0.15 Pain Relevance 0.26
Consequently, unchanged fetal plasma osmolality and sodium levels make it unlikely that the central carbachol induced release of fetal AVP and OT was due to osmotic mechanisms.
Positive_regulation (induced) of Localization (release) of OT in plasma
4) Confidence 0.09 Published 2008 Journal BMC Dev Biol Section Body Doc Link PMC2570685 Disease Relevance 0.37 Pain Relevance 0.03
In addition to hyperosmolar stimulus, hypovolemia and hypotension also can enhance the release of AVP and OT [35,45].
Positive_regulation (enhance) of Localization (release) of OT associated with pressure volume 2 under development
5) Confidence 0.09 Published 2008 Journal BMC Dev Biol Section Body Doc Link PMC2570685 Disease Relevance 0.32 Pain Relevance 0.03
The present study demonstrated, for the first time, that i.c.v. carbachol could induce fetal AVP and OT release at 0.9 gestation, indicating functional maturation of the central cholinergic mechanism-mediated neuroendocrine secretion in the fetal hypothalamus.
Positive_regulation (induce) of Localization (release) of OT in hypothalamus
6) Confidence 0.09 Published 2008 Journal BMC Dev Biol Section Body Doc Link PMC2570685 Disease Relevance 0 Pain Relevance 0.11

General Comments

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