INT193111

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Context Info
Confidence 0.86
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.01
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nuclear chromosome (Birc5) chromosome segregation (Birc5) intracellular (Birc5)
enzyme binding (Birc5) embryo development (Birc5) cytoplasm (Birc5)
Anatomy Link Frequency
poly 1
Birc5 (Mus musculus)
Pain Link Frequency Relevance Heat
cINOD 12 78.80 Quite High
COX-2 inhibitor 3 35.20 Quite Low
Neuropeptide 2 32.84 Quite Low
Neurotransmitter 5 31.84 Quite Low
Inflammation 9 5.00 Very Low Very Low Very Low
cytokine 8 5.00 Very Low Very Low Very Low
Hippocampus 7 5.00 Very Low Very Low Very Low
Eae 6 5.00 Very Low Very Low Very Low
long-term potentiation 5 5.00 Very Low Very Low Very Low
Kinase C 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 178 100.00 Very High Very High Very High
Parkinson's Disease 8 99.68 Very High Very High Very High
Neurodegenerative Disease 36 96.06 Very High Very High Very High
Disease 127 77.88 Quite High
Cancer 87 68.48 Quite High
Death 66 59.92 Quite High
Repression 4 49.12 Quite Low
Colon Cancer 5 42.84 Quite Low
Aneuploidy 1 40.96 Quite Low
Prostate Cancer 3 25.56 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thus, as expected, no correlation between COX-2 expression and the degree of survivin down-regulation by DMC or celecoxib was found.
Protein_catabolism (degree) of survivin
1) Confidence 0.86 Published 2006 Journal Mol Cancer Section Body Doc Link PMC1479836 Disease Relevance 0.14 Pain Relevance 0.08
The proposed basic blueprint for neuronal apoptosis shown in Figure 2 considers a trigger submodule (the DISC-system in the Bentele et al. paper), an actuator (the caspase submodule), a feed-forward module (mitochondrial dysfunction), a brake (IAP/Survivin/Cki) and the many degradative responses of apoptosis (degradation of proteins, poly(ADP-ribose)polymerase (PARP), DNA and so on).
Protein_catabolism (degradation) of Survivin in poly associated with parkinson's disease, apoptosis and neurodegenerative disease
2) Confidence 0.02 Published 2006 Journal BMC Neurosci Section Body Doc Link PMC1775042 Disease Relevance 0.87 Pain Relevance 0

General Comments

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