INT193491

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.36
First Reported 2006
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.67
Pain Relevance 0.40

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Nos3) aging (Nos3) Golgi apparatus (Nos3)
cytoplasm (Nos3) signal transduction (Nos3) oxidoreductase activity (Nos3)
Anatomy Link Frequency
endothelium 2
Nos3 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ischemia 80 99.40 Very High Very High Very High
Bioavailability 2 86.72 High High
cytokine 2 60.28 Quite High
cva 3 11.52 Low Low
medulla 1 7.00 Low Low
Inflammation 7 5.00 Very Low Very Low Very Low
anesthesia 3 5.00 Very Low Very Low Very Low
Inflammatory mediators 3 5.00 Very Low Very Low Very Low
corticosteroid 2 5.00 Very Low Very Low Very Low
halothane 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cv Unclassified Under Development 80 99.40 Very High Very High Very High
Diabetes Mellitus 73 94.48 High High
Hyperhomocysteinemia 4 64.04 Quite High
Urological Neuroanatomy 15 63.00 Quite High
Vasculitis 2 62.64 Quite High
Stress 10 58.64 Quite High
Adhesions 9 56.96 Quite High
Dyslipidemia /

Combined Dyslipidemia

5 53.08 Quite High
Disorder Of Lipid Metabolism 11 51.68 Quite High
Hemorrhage 3 11.52 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These findings suggest that the vascular damage in our animal model is limited to the endothelium per se; however, the stimulated activation of eNOS seems not to be impaired nor does the utilization of the precursor L-arginine.
Negative_regulation (impaired) of Positive_regulation (activation) of eNOS in endothelium
1) Confidence 0.36 Published 2010 Journal Experimental Diabetes Research Section Body Doc Link PMC3014714 Disease Relevance 0.90 Pain Relevance 0.03
Induction of short time (60 min) Isc, in control rats, produced a loss in the glomerular eNOS and an increase of its interstitial expression (c).
Negative_regulation (loss) of Positive_regulation (increase) of eNOS associated with ischemia
2) Confidence 0.34 Published 2006 Journal BMC Nephrol Section Body Doc Link PMC1501003 Disease Relevance 0.71 Pain Relevance 0.37

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox