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Context Info
Confidence 0.69
First Reported 1991
Last Reported 2010
Negated 0
Speculated 1
Reported most in Abstract
Documents 2
Total Number 3
Disease Relevance 1.40
Pain Relevance 0.32

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (AGXT) small molecule metabolic process (AGXT) peroxisome (AGXT)
cellular nitrogen compound metabolic process (AGXT)
AGXT (Homo sapiens)
Pain Link Frequency Relevance Heat
Hsan 4 99.36 Very High Very High Very High
headache 1 92.20 High High
Pain 4 50.00 Quite Low
analgesia 1 50.00 Quite Low
Acupuncture 1 50.00 Quite Low
Central nervous system 3 5.00 Very Low Very Low Very Low
Analgesic 1 5.00 Very Low Very Low Very Low
Pain threshold 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Anaemia 1 99.88 Very High Very High Very High
Thrombocytopenia 1 99.72 Very High Very High Very High
Hereditary Sensory And Autonomic Neuropathies 4 99.36 Very High Very High Very High
Leukopenia 1 99.36 Very High Very High Very High
Congenital Anomalies 1 98.98 Very High Very High Very High
Exanthema 1 95.00 High High
Appetite Loss 1 92.96 High High
Myalgia 1 92.56 High High
Headache 1 92.20 High High
Fever 1 91.48 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Commonly observed laboratory abnormalities include leukopenia, thrombocytopenia, anemia, and elevated hepatic aminotransferase levels.
Positive_regulation (elevated) of hepatic aminotransferase associated with anaemia, leukopenia, congenital anomalies and thrombocytopenia
1) Confidence 0.69 Published 1991 Journal Infect. Dis. Clin. North Am. Section Abstract Doc Link 2051014 Disease Relevance 1.11 Pain Relevance 0.09
MA also increased SPT and TST, but the changes were not significantly different from those induced by SA.
Positive_regulation (increased) of SPT
2) Confidence 0.67 Published 2010 Journal Eur J Pain Section Body Doc Link 20045360 Disease Relevance 0.05 Pain Relevance 0
Furthermore, mutations in this lysine and in a histidine residue that is also predicted to be important for pyridoxal 5'-phosphate binding to Lcb2p also dominantly inactivate SPT similar to the hereditary sensory neuropathy type 1-like mutations in Lcb1p.
Spec (like) Positive_regulation (inactivate) of SPT associated with hsan
3) Confidence 0.02 Published 2002 Journal J. Biol. Chem. Section Abstract Doc Link 11781309 Disease Relevance 0.23 Pain Relevance 0.23

General Comments

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