INT193810

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Context Info
Confidence 0.27
First Reported 2006
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.13
Pain Relevance 0.20

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

small molecule metabolic process (APOE) aging (APOE) Golgi apparatus (APOE)
lipid metabolic process (APOE) cytoplasm (APOE) lipid binding (APOE)
APOE (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 4 90.80 High High
chemokine 9 80.88 Quite High
metalloproteinase 2 68.88 Quite High
antagonist 14 59.04 Quite High
cerebral cortex 2 54.48 Quite High
Glutamate 24 5.00 Very Low Very Low Very Low
amygdala 13 5.00 Very Low Very Low Very Low
nMDA receptor 12 5.00 Very Low Very Low Very Low
Opioid 7 5.00 Very Low Very Low Very Low
Dopamine 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 17 97.28 Very High Very High Very High
Toxicity 43 93.80 High High
Alzheimer's Dementia 11 92.88 High High
INFLAMMATION 4 90.80 High High
Acquired Immune Deficiency Syndrome Or Hiv Infection 89 89.36 High High
Drug Induced Neurotoxicity 38 75.52 Quite High
Dementia 23 74.08 Quite High
Disease 75 68.16 Quite High
Amyloidosis 8 50.00 Quite Low
Feminization 1 25.28 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Also Tat and apoE compete for the same binding site, resulting in increased extracellular Tat; the ability for apoE to
Phosphorylation (ability) of apoE
1) Confidence 0.27 Published 2006 Journal Journal of Biomedicine and Biotechnology Section Body Doc Link PMC1510947 Disease Relevance 0.79 Pain Relevance 0.13
The lack of apo-E in the offspring of ?
Phosphorylation (lack) of apo-E
2) Confidence 0.11 Published 2007 Journal Acta Neuropathol Section Body Doc Link PMC1824787 Disease Relevance 0.34 Pain Relevance 0.07

General Comments

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