INT193955

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Context Info
Confidence 0.37
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.64
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (Psen1, Psen2) endoplasmic reticulum (Psen1, Psen2) embryo development (Psen1, Psen2)
protein complex (Psen1, Psen2) peptidase activity (Psen1, Psen2) nucleus (Psen1, Psen2)
Psen1 (Mus musculus)
Psen2 (Mus musculus)
Pain Link Frequency Relevance Heat
cINOD 4 45.96 Quite Low
sodium channel 2 5.00 Very Low Very Low Very Low
Eae 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 22 99.84 Very High Very High Very High
Disease 126 99.16 Very High Very High Very High
Disease Progression 2 83.76 Quite High
INFLAMMATION 2 23.20 Low Low
Alzheimer's Dementia 12 5.00 Very Low Very Low Very Low
Apoptosis 10 5.00 Very Low Very Low Very Low
Adhesions 10 5.00 Very Low Very Low Very Low
Frontotemporal Dementia 10 5.00 Very Low Very Low Very Low
Urological Neuroanatomy 2 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The lesser frequency of FAD-linked PS2 mutations compared to that of PS1 also suggests a redundant role for PS2 containing ?
PS1 Binding (role) of PS2 associated with disease
1) Confidence 0.37 Published 2006 Journal Mol Neurodegener Section Body Doc Link PMC1513131 Disease Relevance 0.85 Pain Relevance 0
Indeed, in vivo complementation of PS1 deficiency with PS2 in transgenic mice indicates functionally different roles for PS1 and PS2 [70].
PS1 Binding (complementation) of PS2 associated with targeted disruption
2) Confidence 0.37 Published 2006 Journal Mol Neurodegener Section Body Doc Link PMC1513131 Disease Relevance 0.79 Pain Relevance 0

General Comments

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