INT193962

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Context Info
Confidence 0.13
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.08
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (Psen1, Ncstn) endoplasmic reticulum (Psen1, Ncstn) peptidase activity (Psen1, Ncstn)
plasma membrane (Psen1, Ncstn) mitochondrion (Psen1) intracellular (Psen1)
Psen1 (Mus musculus)
Ncstn (Mus musculus)
Pain Link Frequency Relevance Heat
sodium channel 2 39.16 Quite Low
cINOD 4 5.00 Very Low Very Low Very Low
Eae 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 22 77.12 Quite High
Adhesions 10 31.08 Quite Low
Disease 126 14.28 Low Low
Alzheimer's Dementia 12 5.00 Very Low Very Low Very Low
Apoptosis 10 5.00 Very Low Very Low Very Low
Frontotemporal Dementia 10 5.00 Very Low Very Low Very Low
Urological Neuroanatomy 2 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Disease Progression 2 5.00 Very Low Very Low Very Low
INFLAMMATION 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
-secretase inhibitors markedly increase cell surface levels of PS1 and PEN-2 without affecting that of nicastrin also implies a tighter association between PS1 and PEN-2 [61].
PS1 Binding (association) of nicastrin
1) Confidence 0.13 Published 2006 Journal Mol Neurodegener Section Body Doc Link PMC1513131 Disease Relevance 0 Pain Relevance 0
A type I transmembrane protein termed Nicastrin, which is the mammalian homolog of aph-2, was identified simultaneously by biochemical methods as a protein bound to PS1 [54].
PS1 Binding (bound) of Nicastrin
2) Confidence 0.13 Published 2006 Journal Mol Neurodegener Section Body Doc Link PMC1513131 Disease Relevance 0.08 Pain Relevance 0

General Comments

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