INT195015

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Context Info
Confidence 0.04
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.31
Pain Relevance 0.15

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Slc8a1) plasma membrane (Slc8a1) transmembrane transport (Slc8a1)
response to stress (Slc8a1)
Anatomy Link Frequency
neuronal 2
Slc8a1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Glutamate 144 97.96 Very High Very High Very High
nMDA receptor 6 75.60 Quite High
ischemia 3 5.00 Very Low Very Low Very Low
Neurotransmitter 3 5.00 Very Low Very Low Very Low
excitatory amino acid 1 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
Glutamate receptor 1 5.00 Very Low Very Low Very Low
Neuritis 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 97 85.12 High High
Death 80 83.92 Quite High
Toxicity 3 50.00 Quite Low
Injury 2 6.88 Low Low
Neurodegenerative Disease 11 5.00 Very Low Very Low Very Low
Aging 3 5.00 Very Low Very Low Very Low
Disease 3 5.00 Very Low Very Low Very Low
Cv General 4 Under Development 2 5.00 Very Low Very Low Very Low
Neuritis 1 5.00 Very Low Very Low Very Low
Parkinson's Disease 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Whether glutamate induced Ca2+ influx and the activation of calpain observed in our neuronal cells also results in the inactivation of NCX that can be prevented by estrogen remains to be investigated.
Negative_regulation (prevented) of Positive_regulation (inactivation) of NCX in neuronal associated with glutamate
1) Confidence 0.04 Published 2006 Journal BMC Neurosci Section Body Doc Link PMC1526740 Disease Relevance 0.31 Pain Relevance 0.15

General Comments

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