INT195429

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Context Info
Confidence 0.42
First Reported 2006
Last Reported 2006
Negated 2
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.01
Pain Relevance 1.98

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (SLC6A2) plasma membrane (SLC6A2) transmembrane transport (SLC6A2)
Anatomy Link Frequency
synapse 4
SLC6A2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Serotonin 10 100.00 Very High Very High Very High
monoamine 42 99.96 Very High Very High Very High
Neurotransmitter 40 99.56 Very High Very High Very High
Pain 176 96.96 Very High Very High Very High
Analgesic 10 75.44 Quite High
visual analogue scale 8 5.00 Very Low Very Low Very Low
Dopamine 8 5.00 Very Low Very Low Very Low
Catecholamine 6 5.00 Very Low Very Low Very Low
Acute pain 6 5.00 Very Low Very Low Very Low
tolerance 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pain 184 88.80 High High
Targeted Disruption 2 40.32 Quite Low
Aggression 2 39.12 Quite Low
Nociception 6 18.96 Low Low
Hypersensitivity 2 7.84 Low Low
Disease 4 5.00 Very Low Very Low Very Low
Impacted Tooth 2 5.00 Very Low Very Low Very Low
Neck Pain 2 5.00 Very Low Very Low Very Low
Cognitive Disorder 2 5.00 Very Low Very Low Very Low
Temporomandibular Joint Disorders 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Since the re-uptake of monoamine neurotransmitters through specific transporters is the major method of elimination from the neural synapse, it is not surprising that the genetic variations in norepinephrine transporter gene (SLC6A2) and the serotonin transporter gene (SLC6A4) affect monoamine neurotransmitter mediated human behaviors including responses to painful stimuli.
Neg (not) Regulation (affect) of Positive_regulation (variations) of norepinephrine transporter in synapse associated with pain, neurotransmitter, serotonin and monoamine
1) Confidence 0.42 Published 2006 Journal Mol Pain Section Body Doc Link PMC1543620 Disease Relevance 0.50 Pain Relevance 0.99
Since the re-uptake of monoamine neurotransmitters through specific transporters is the major method of elimination from the neural synapse, it is not surprising that the genetic variations in norepinephrine transporter gene (SLC6A2) and the serotonin transporter gene (SLC6A4) affect monoamine neurotransmitter mediated human behaviors including responses to painful stimuli.
Neg (not) Regulation (affect) of Positive_regulation (variations) of SLC6A2 in synapse associated with pain, neurotransmitter, serotonin and monoamine
2) Confidence 0.42 Published 2006 Journal Mol Pain Section Body Doc Link PMC1543620 Disease Relevance 0.50 Pain Relevance 0.99

General Comments

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