INT195454

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Context Info
Confidence 0.45
First Reported 2006
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 5
Total Number 7
Disease Relevance 7.65
Pain Relevance 0.85

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (SAT1) small molecule metabolic process (SAT1) intracellular (SAT1)
transferase activity, transferring acyl groups (SAT1) cellular nitrogen compound metabolic process (SAT1) cytoplasm (SAT1)
SAT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Endocannabinoid 140 100.00 Very High Very High Very High
agonist 8 79.44 Quite High
Cannabinoid receptor 28 79.08 Quite High
Potency 4 75.08 Quite High
Inflammation 23 5.00 Very Low Very Low Very Low
alcohol 6 5.00 Very Low Very Low Very Low
lidocaine 4 5.00 Very Low Very Low Very Low
Cannabinoid 4 5.00 Very Low Very Low Very Low
cINOD 3 5.00 Very Low Very Low Very Low
cytokine 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Obesity 1006 100.00 Very High Very High Very High
Diabetes Mellitus 140 99.18 Very High Very High Very High
Prostate Cancer 1 97.96 Very High Very High Very High
Agammaglobulinemia 2 94.64 High High
Breast Cancer 1 94.00 High High
Cancer 29 87.00 High High
Colorectal Cancer 23 82.24 Quite High
Carcinoma 1 79.12 Quite High
Colon Cancer 3 75.20 Quite High
Disorder Of Lipid Metabolism 106 74.88 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
On the other hand, in prostatic cancer the SSAT overexpression induces growth inhibition without determining polyamine pool depletion since SSAT activity seems to modulate ODC activity [11,34].
Spec (seems) Regulation (modulate) of SSAT associated with prostate cancer
1) Confidence 0.45 Published 2006 Journal BMC Cancer Section Body Doc Link PMC1543652 Disease Relevance 0.80 Pain Relevance 0
Possible explanations for the observed differential alterations of endocannabinoid levels in the SAT of OBT2D patients
Regulation (alterations) of SAT associated with endocannabinoid and obesity
2) Confidence 0.12 Published 2010 Journal Lipids Health Dis Section Body Doc Link PMC2868848 Disease Relevance 0.61 Pain Relevance 0.26
Possible significance of the observed alterations of the concentrations of endocannabinoids and related lipids in the SAT of OBT2D patients
Regulation (alterations) of SAT associated with endocannabinoid and obesity
3) Confidence 0.12 Published 2010 Journal Lipids Health Dis Section Body Doc Link PMC2868848 Disease Relevance 1.26 Pain Relevance 0.23
However, we have also found that, in the presence of T2D, obese patients do exhibit differential alterations of the SAT concentrations of the four measured lipid mediators.
Regulation (alterations) of SAT associated with diabetes mellitus and obesity
4) Confidence 0.12 Published 2010 Journal Lipids Health Dis Section Body Doc Link PMC2868848 Disease Relevance 1.19 Pain Relevance 0.31
As shown in Fig. 1, the SAT concentrations of AEA, OEA and PEA were significantly higher in OBT2D than in either OB or NW subjects.
Regulation (concentrations) of SAT associated with obesity
5) Confidence 0.07 Published 2010 Journal Lipids Health Dis Section Body Doc Link PMC2868848 Disease Relevance 1.25 Pain Relevance 0.05
Our data thus suggest that some genes in SAT 4 and SAT 8 are involved in immune-related signalling pathways such as the Toll-like receptor signalling and the Fc?
Regulation (involved) of SAT associated with obesity
6) Confidence 0.06 Published 2010 Journal BMC Med Genomics Section Body Doc Link PMC2925326 Disease Relevance 1.27 Pain Relevance 0
Our data thus suggest that some genes in SAT 4 and SAT 8 are involved in immune-related signalling pathways such as the Toll-like receptor signalling and the Fc?
Regulation (involved) of SAT 8 associated with obesity
7) Confidence 0.06 Published 2010 Journal BMC Med Genomics Section Body Doc Link PMC2925326 Disease Relevance 1.27 Pain Relevance 0

General Comments

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