INT195927

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Context Info
Confidence 0.07
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.28
Pain Relevance 0.92

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Prkaca, Trpv1) transport (Trpv1) mitochondrion (Prkaca)
Golgi apparatus (Prkaca) protein complex (Prkaca) transmembrane transport (Trpv1)
Prkaca (Mus musculus)
Trpv1 (Mus musculus)
Pain Link Frequency Relevance Heat
qutenza 68 98.44 Very High Very High Very High
Hyperalgesia 9 95.72 Very High Very High Very High
Inflammation 32 95.20 Very High Very High Very High
IPN 4 91.92 High High
Morphine 53 75.00 Quite High
MU agonist 3 75.00 Quite High
opioid receptor 110 74.48 Quite High
agonist 14 72.72 Quite High
Opioid 18 5.00 Very Low Very Low Very Low
Kinase C 14 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hyperalgesia 12 95.72 Very High Very High Very High
INFLAMMATION 31 95.20 Very High Very High Very High
Inflammatory Pain 4 91.92 High High
Nociception 10 25.00 Low Low
Ganglion Cysts 9 25.00 Low Low
Neuropathic Pain 4 5.00 Very Low Very Low Very Low
Neurogenic Inflammation 2 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Diabetic Neuropathy 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Several receptors including G-protein coupled prostaglandin receptors have been reported to functionally interact with the TRPV1 through a cAMP-dependent protein kinase A (PKA) pathway to potentiate TRPV1-mediated capsaicin responses.
PKA Binding (interact) of TRPV1 associated with qutenza
1) Confidence 0.07 Published 2006 Journal Mol Pain Section Abstract Doc Link PMC1553434 Disease Relevance 0.28 Pain Relevance 0.92

General Comments

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