INT195929

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Context Info
Confidence 0.02
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.33
Pain Relevance 1.27

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (OPRM1, Prkaca) plasma membrane (OPRM1, Prkaca) mitochondrion (Prkaca)
endoplasmic reticulum (OPRM1) protein complex (Prkaca) cytoplasm (Prkaca)
OPRM1 (Homo sapiens)
Prkaca (Mus musculus)
Pain Link Frequency Relevance Heat
opioid receptor 110 100.00 Very High Very High Very High
Morphine 53 99.48 Very High Very High Very High
qutenza 68 98.08 Very High Very High Very High
Opioid 18 88.48 High High
Inflammation 32 79.00 Quite High
IPN 4 74.88 Quite High
agonist 14 64.52 Quite High
Cannabinoid receptor 1 58.92 Quite High
Hyperalgesia 9 50.72 Quite High
MU agonist 3 50.00 Quite Low
Disease Link Frequency Relevance Heat
INFLAMMATION 31 79.00 Quite High
Inflammatory Pain 4 74.88 Quite High
Hyperalgesia 12 50.72 Quite High
Neuropathic Pain 4 40.24 Quite Low
Nociception 10 36.04 Quite Low
Ganglion Cysts 9 5.00 Very Low Very Low Very Low
Neurogenic Inflammation 2 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Diabetic Neuropathy 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The results presented here support the hypothesis that the MOP and TRPV1 can functionally interact through a cAMP-dependent PKA pathway, as morphine inhibited FSK-potentiated capsaicin responses but not capsaicin responses in the absence of FSK.
MOP Binding (interact) of PKA associated with qutenza, opioid receptor and morphine
1) Confidence 0.02 Published 2006 Journal Mol Pain Section Body Doc Link PMC1553434 Disease Relevance 0.33 Pain Relevance 1.27

General Comments

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