INT196810

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Context Info
Confidence 0.14
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.07

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Prkaca, Ppp2ca) nucleus (Prkaca, Ppp2ca) plasma membrane (Prkaca, Ppp2ca)
mitochondrion (Prkaca) Golgi apparatus (Prkaca) protein complex (Prkaca)
Prkaca (Mus musculus)
Ppp2ca (Mus musculus)
Pain Link Frequency Relevance Heat
Dopamine 119 96.48 Very High Very High Very High
Action potential 4 44.68 Quite Low
Glutamate 31 5.00 Very Low Very Low Very Low
long-term potentiation 8 5.00 Very Low Very Low Very Low
projection neuron 4 5.00 Very Low Very Low Very Low
Neurotransmitter 3 5.00 Very Low Very Low Very Low
imagery 3 5.00 Very Low Very Low Very Low
nMDA receptor 2 5.00 Very Low Very Low Very Low
Calcium channel 2 5.00 Very Low Very Low Very Low
dopamine receptor 2 5.00 Very Low Very Low Very Low

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Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The main findings from these simulations suggest that the presence of the PKA–PP2A–phosphoThr75 loop causes increased formation of free PKAc as well as phosphoThr34 if a dopamine input is paired with a transient calcium elevation.
PKA Binding (presence) of PP2A associated with dopamine
1) Confidence 0.14 Published 2006 Journal PLoS Computational Biology Section Body Doc Link PMC1562452 Disease Relevance 0 Pain Relevance 0.07

General Comments

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