INT196877

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Context Info
Confidence 0.11
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.41
Pain Relevance 0.62

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Trpv1) intracellular (Calca) cytoplasm (Calca)
transmembrane transport (Trpv1) lipid metabolic process (Trpv1) cytosol (Trpv1)
Anatomy Link Frequency
heart 2
Trpv1 (Mus musculus)
Calca (Mus musculus)
Pain Link Frequency Relevance Heat
Calcitonin gene-related peptide 7 100.00 Very High Very High Very High
ischemia 5 98.72 Very High Very High Very High
antagonist 2 98.24 Very High Very High Very High
substance P 5 93.72 High High
Pain 6 92.88 High High
qutenza 2 60.08 Quite High
c fibre 1 59.16 Quite High
agonist 2 53.24 Quite High
nociceptor 6 50.00 Quite Low
Kinase C 8 27.48 Quite Low
Disease Link Frequency Relevance Heat
Targeted Disruption 3 99.38 Very High Very High Very High
Coronary Artery Disease 2 98.72 Very High Very High Very High
Nociception 21 87.52 High High
Cv Unclassified Under Development 3 68.72 Quite High
Coronary Heart Disease 1 53.04 Quite High
Diabetes Mellitus 1 45.04 Quite Low
Pain 5 34.96 Quite Low
INFLAMMATION 5 11.24 Low Low
Increased Venous Pressure Under Development 5 5.00 Very Low Very Low Very Low
Injury 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This possibility is supported by the finding that recovery from myocardial ischemia is compromised in TRPV1 knockout mice [28] and proton mediated CGRP release from the heart is mediated exclusively by TRPV1 [29,30].
TRPV1 Positive_regulation (mediated) of Localization (release) of CGRP in heart associated with targeted disruption, coronary artery disease, ischemia and calcitonin gene-related peptide
1) Confidence 0.11 Published 2006 Journal Mol Pain Section Body Doc Link PMC1563450 Disease Relevance 0.41 Pain Relevance 0.62

General Comments

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