INT197212

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.35
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Title
Documents 1
Total Number 1
Disease Relevance 0.10
Pain Relevance 0.43

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Kars) translation (Kars) ligase activity (Kars)
cytoplasm (Kars)
Anatomy Link Frequency
spinal 1
Kars (Mus musculus)
Pain Link Frequency Relevance Heat
substantia gelatinosa 57 100.00 Very High Very High Very High
GABAergic 13 94.64 High High
Dorsal horn neuron 5 92.24 High High
agonist 4 75.52 Quite High
Action potential 5 57.16 Quite High
antagonist 8 43.44 Quite Low
Spinal cord 18 5.00 Very Low Very Low Very Low
gABA 12 5.00 Very Low Very Low Very Low
Dorsal horn 11 5.00 Very Low Very Low Very Low
spinal dorsal horn 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Nociception 7 98.80 Very High Very High Very High
Targeted Disruption 4 5.00 Very Low Very Low Very Low
Pain 3 5.00 Very Low Very Low Very Low
Epilepsy 1 5.00 Very Low Very Low Very Low
Overdose 1 5.00 Very Low Very Low Very Low
Congenital Anomalies 1 5.00 Very Low Very Low Very Low
Anxiety Disorder 1 5.00 Very Low Very Low Very Low
Inflammatory Pain 1 5.00 Very Low Very Low Very Low
INFLAMMATION 1 5.00 Very Low Very Low Very Low
Disseminated Intravascular Coagulation 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Presynaptic regulation of the inhibitory transmission by GluR5-containing kainate receptors in spinal substantia gelatinosa

GluR5-containing kainate receptors (KARs) are known to be involved in nociceptive transmission.

Regulation (regulation) of KARs in spinal associated with nociception and substantia gelatinosa
1) Confidence 0.35 Published 2006 Journal Mol Pain Section Title Doc Link PMC1570342 Disease Relevance 0.10 Pain Relevance 0.43

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox