INT197227

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Context Info
Confidence 0.06
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.08
Pain Relevance 0.46

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Rora) DNA binding (Rora)
Anatomy Link Frequency
spinal 1
Rora (Mus musculus)
Pain Link Frequency Relevance Heat
substantia gelatinosa 57 100.00 Very High Very High Very High
agonist 4 96.16 Very High Very High Very High
Dorsal horn neuron 5 87.84 High High
GABAergic 13 85.04 High High
Action potential 5 59.44 Quite High
antagonist 8 32.00 Quite Low
Spinal cord 18 5.00 Very Low Very Low Very Low
gABA 12 5.00 Very Low Very Low Very Low
Dorsal horn 11 5.00 Very Low Very Low Very Low
spinal dorsal horn 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Nociception 7 80.96 Quite High
Targeted Disruption 4 5.00 Very Low Very Low Very Low
Pain 3 5.00 Very Low Very Low Very Low
Epilepsy 1 5.00 Very Low Very Low Very Low
Overdose 1 5.00 Very Low Very Low Very Low
Congenital Anomalies 1 5.00 Very Low Very Low Very Low
Anxiety Disorder 1 5.00 Very Low Very Low Very Low
Inflammatory Pain 1 5.00 Very Low Very Low Very Low
INFLAMMATION 1 5.00 Very Low Very Low Very Low
Disseminated Intravascular Coagulation 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In the present study, pharmacological, electrophysiological and genetic methods were used to show that presynaptic GluR5 KARs are involved in the modulation of inhibitory transmission in the SG of spinal slices in vitro.
Regulation (modulation) of SG in spinal associated with substantia gelatinosa
1) Confidence 0.06 Published 2006 Journal Mol Pain Section Abstract Doc Link PMC1570342 Disease Relevance 0.08 Pain Relevance 0.46

General Comments

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