INT197432

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Context Info
Confidence 0.29
First Reported 2006
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 0.85
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (COL4A1)
COL4A1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 3 23.60 Low Low
Inflammatory response 2 18.80 Low Low
metalloproteinase 6 5.00 Very Low Very Low Very Low
fibrosis 3 5.00 Very Low Very Low Very Low
agonist 3 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
cOX1 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
Mechanotransduction 1 5.00 Very Low Very Low Very Low
pain pelvic 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Uterine Fibroids 120 99.64 Very High Very High Very High
Fracture Healing 64 93.04 High High
Osteoporosis 3 51.64 Quite High
INFLAMMATION 5 23.60 Low Low
Rheumatoid Arthritis 24 21.32 Low Low
Adhesions 2 9.20 Low Low
Cancer 9 5.00 Very Low Very Low Very Low
Injury 3 5.00 Very Low Very Low Very Low
Papillomavirus Infection 3 5.00 Very Low Very Low Very Low
Apoptosis 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
At higher concentrations of ATRA, the relative COL4A1 gene expression between leiomyoma and myometrial cells remained comparable.
Positive_regulation (concentrations) of Gene_expression (expression) of COL4A1 gene associated with uterine fibroids
1) Confidence 0.29 Published 2008 Journal Clinical Endocrinology Section Body Doc Link PMC2610401 Disease Relevance 0.64 Pain Relevance 0
COL3A1 and COL4A1 were shown to be overexpressed in the PF7 and PF10 ESTs (Table 2).
Positive_regulation (overexpressed) of Gene_expression (overexpressed) of COL4A1
2) Confidence 0.14 Published 2006 Journal BMC Genomics Section Body Doc Link PMC1578570 Disease Relevance 0.21 Pain Relevance 0

General Comments

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