INT199103

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Context Info
Confidence 0.05
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.09
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Akt1, Ncf2) cytosol (Akt1, Ncf2) transport (Akt1)
aging (Akt1) enzyme binding (Akt1) carbohydrate metabolic process (Akt1)
Akt1 (Mus musculus)
Ncf2 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammatory stimuli 2 85.84 High High
Kinase C 1 38.04 Quite Low
cytokine 5 21.92 Low Low
Inflammation 10 5.00 Very Low Very Low Very Low
cINOD 6 5.00 Very Low Very Low Very Low
Inflammatory response 5 5.00 Very Low Very Low Very Low
chemokine 2 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
Nerve growth factor 1 5.00 Very Low Very Low Very Low
ischemia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 19 85.48 High High
Stress 3 50.00 Quite Low
Disease 58 5.00 Very Low Very Low Very Low
Death 8 5.00 Very Low Very Low Very Low
Amyloid Plaque 7 5.00 Very Low Very Low Very Low
Cognitive Disorder 6 5.00 Very Low Very Low Very Low
Alzheimer's Dementia 6 5.00 Very Low Very Low Very Low
Drug Induced Neurotoxicity 5 5.00 Very Low Very Low Very Low
Neurodegenerative Disease 4 5.00 Very Low Very Low Very Low
Neutrophil Disorders 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
While the precise role of Rac in NADPH oxidase activation is not fully understood, several lines of evidence suggest Rac acts as an adaptor molecule for p67phox [46,47], and may be actively involved in the electron transfer process itself [48,49].
Rac Positive_regulation (molecule) of p67phox
1) Confidence 0.05 Published 2006 Journal J Neuroinflammation Section Body Doc Link PMC1637099 Disease Relevance 0.09 Pain Relevance 0.04

General Comments

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