INT199108

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Context Info
Confidence 0.02
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.07
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Akt1, Arhgdia) transport (Akt1) aging (Akt1)
enzyme binding (Akt1) carbohydrate metabolic process (Akt1) cytosol (Akt1)
Akt1 (Mus musculus)
Arhgdia (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammatory stimuli 2 75.20 Quite High
cytokine 5 43.92 Quite Low
Kinase C 1 27.40 Quite Low
Inflammation 10 5.00 Very Low Very Low Very Low
cINOD 6 5.00 Very Low Very Low Very Low
Inflammatory response 5 5.00 Very Low Very Low Very Low
chemokine 2 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
Nerve growth factor 1 5.00 Very Low Very Low Very Low
ischemia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 19 74.84 Quite High
Stress 3 50.00 Quite Low
Disease 58 5.00 Very Low Very Low Very Low
Death 8 5.00 Very Low Very Low Very Low
Amyloid Plaque 7 5.00 Very Low Very Low Very Low
Cognitive Disorder 6 5.00 Very Low Very Low Very Low
Alzheimer's Dementia 6 5.00 Very Low Very Low Very Low
Drug Induced Neurotoxicity 5 5.00 Very Low Very Low Very Low
Neurodegenerative Disease 4 5.00 Very Low Very Low Very Low
Neutrophil Disorders 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The requirement for Rac-GTP has been further established through in vitro studies demonstrating that the addition of dominant-negative Rac or overexpression of Rho-GDI severely diminishes superoxide production [50,51].


Rac Negative_regulation (diminishes) of Gene_expression (overexpression) of Rho-GDI
1) Confidence 0.02 Published 2006 Journal J Neuroinflammation Section Body Doc Link PMC1637099 Disease Relevance 0.07 Pain Relevance 0.04

General Comments

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