INT200647

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Context Info
Confidence 0.10
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.93
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (HDAC4, CASP9) nucleus (HDAC4, CASP9) peptidase activity (CASP9)
intracellular (CASP9) transcription factor binding (HDAC4) cytoplasm (HDAC4)
Anatomy Link Frequency
apoptotic cell 1
HDAC4 (Homo sapiens)
CASP9 (Homo sapiens)
Pain Link Frequency Relevance Heat
Migraine 1 5.00 Very Low Very Low Very Low
withdrawal 1 5.00 Very Low Very Low Very Low
headache 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 41 100.00 Very High Very High Very High
Death 25 100.00 Very High Very High Very High
Ovarian Cancer 2 83.76 Quite High
Cancer 26 81.84 Quite High
Cervical Cancer 5 5.00 Very Low Very Low Very Low
Repression 2 5.00 Very Low Very Low Very Low
Malignant Neoplastic Disease 2 5.00 Very Low Very Low Very Low
Epilepsy 1 5.00 Very Low Very Low Very Low
Headache 1 5.00 Very Low Very Low Very Low
Cluster Headache 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Taken together, our data suggest that the HDAC inhibitor-induced apoptotic cell death is attained by activating both the caspase-8 and caspase-9 activities.


HDAC Positive_regulation (activating) of caspase-9 in apoptotic cell associated with apoptosis and death
1) Confidence 0.10 Published 2006 Journal Mol Cancer Section Body Doc Link PMC1762018 Disease Relevance 0.93 Pain Relevance 0

General Comments

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