INT200676

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Context Info
Confidence 0.53
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.01
Pain Relevance 0.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (AKT2) transport (AKT2) mitochondrion (AKT2)
carbohydrate metabolic process (AKT2) cytoplasm (AKT2) cytosol (AKT2)
AKT2 (Homo sapiens)
Pain Link Frequency Relevance Heat
withdrawal 2 85.44 High High
headache 2 55.20 Quite High
Migraine 2 54.44 Quite High
Disease Link Frequency Relevance Heat
Apoptosis 82 91.12 High High
Cancer 52 72.20 Quite High
Convulsion 2 57.00 Quite High
Cluster Headache 2 55.20 Quite High
Headache 2 54.44 Quite High
Epilepsy 2 54.04 Quite High
Malignant Neoplastic Disease 4 35.24 Quite Low
Repression 4 34.80 Quite Low
Death 50 5.00 Very Low Very Low Very Low
Cervical Cancer 10 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The phosphorylation of a conserved threonine residue (Thr308 in Akt1, Thr309 in Akt2 and Thr305 in Akt3), upon growth factor stimulation, is required for Akt activation, while the phosphorylation of a serine residue (Ser473 in Akt1, Ser474 in Akt2 and Ser474 in Akt3) is only required for maximal Akt activity [21,24-26].
Phosphorylation (phosphorylation) of Akt2
1) Confidence 0.53 Published 2006 Journal Mol Cancer Section Body Doc Link PMC1762018 Disease Relevance 0.43 Pain Relevance 0.04
The phosphorylation of a conserved threonine residue (Thr308 in Akt1, Thr309 in Akt2 and Thr305 in Akt3), upon growth factor stimulation, is required for Akt activation, while the phosphorylation of a serine residue (Ser473 in Akt1, Ser474 in Akt2 and Ser474 in Akt3) is only required for maximal Akt activity [21,24-26].
Phosphorylation (phosphorylation) of Akt2
2) Confidence 0.18 Published 2006 Journal Mol Cancer Section Body Doc Link PMC1762018 Disease Relevance 0.58 Pain Relevance 0.15

General Comments

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