INT201139

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Context Info
Confidence 0.73
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.15
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Sox2) anatomical structure formation involved in morphogenesis (Sox2) nucleus (Sox2)
DNA binding (Sox2) transcription factor binding (Sox2) cytoplasm (Sox2)
Sox2 (Mus musculus)
Pain Link Frequency Relevance Heat
imagery 9 5.00 Very Low Very Low Very Low
Calcium channel 2 5.00 Very Low Very Low Very Low
tetrodotoxin 1 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
fibrosis 1 5.00 Very Low Very Low Very Low
depression 1 5.00 Very Low Very Low Very Low
addiction 1 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
tolerance 1 5.00 Very Low Very Low Very Low
Action potential 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hypertrophy 80 76.76 Quite High
Myocardial Infarction 27 35.40 Quite Low
Cv Unclassified Under Development 5 16.64 Low Low
Body Weight 1 8.64 Low Low
Coronary Heart Disease 3 6.04 Low Low
Hypertension 2 5.00 Very Low Very Low Very Low
Fibrosis 1 5.00 Very Low Very Low Very Low
Dilated Cardiomyopathy 1 5.00 Very Low Very Low Very Low
Depression 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, with further decrease of LCC-RyR coupling efficiency, an increasing fraction of CRUs tends not to be activated before ICa inactivation (Figure 5B; green, blue, and black lines in Figure 6C); global performance of Ca2+ release declines.
Localization (decrease) of LCC
1) Confidence 0.73 Published 2007 Journal PLoS Biology Section Body Doc Link PMC1764437 Disease Relevance 0.15 Pain Relevance 0

General Comments

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