INT201484

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.34
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.96
Pain Relevance 0.32

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Prkcg, Ednrb) aging (Ednrb) intracellular (Prkcg)
signal transducer activity (Ednrb) response to stress (Prkcg) cytoplasm (Prkcg)
Prkcg (Rattus norvegicus)
Ednrb (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Kinase C 25 100.00 Very High Very High Very High
cva 4 94.60 High High
ischemia 10 50.00 Quite Low
agonist 2 5.00 Very Low Very Low Very Low
halothane 2 5.00 Very Low Very Low Very Low
withdrawal 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stroke 5 97.36 Very High Very High Very High
Cv General 4 Under Development 14 94.60 High High
Middle Cerebral Artery Infarction 15 92.56 High High
Myocardial Infarction 1 67.00 Quite High
Cardiovascular Disease 1 65.68 Quite High
Infarction 9 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 2 5.00 Very Low Very Low Very Low
Weight Loss 1 5.00 Very Low Very Low Very Low
Cv Unclassified Under Development 1 5.00 Very Low Very Low Very Low
Disease 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The aim of the present study was to examine if a general PKC inhibitor, Ro-32-0432, can decrease the ETB receptor upregulation in MCA and reduce the ischemic infarct volume after experimental ischemic stroke.
Negative_regulation (inhibitor) of PKC Negative_regulation (decrease) of Positive_regulation (upregulation) of ETB receptor associated with kinase c and stroke
1) Confidence 0.34 Published 2007 Journal BMC Neurosci Section Body Doc Link PMC1770924 Disease Relevance 0.96 Pain Relevance 0.32

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox