INT201513

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Context Info
Confidence 0.50
First Reported 2007
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 2.63
Pain Relevance 0.16

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

aging (FAS) protein complex assembly (FAS) cytoplasm (FAS)
signal transducer activity (FAS) cytosol (FAS) signal transduction (FAS)
Anatomy Link Frequency
hepatocytes 2
FAS (Homo sapiens)
Pain Link Frequency Relevance Heat
imagery 18 87.28 High High
anesthesia 1 57.48 Quite High
Inflammation 48 5.00 Very Low Very Low Very Low
cytokine 21 5.00 Very Low Very Low Very Low
fibrosis 19 5.00 Very Low Very Low Very Low
Inflammatory response 11 5.00 Very Low Very Low Very Low
insula 6 5.00 Very Low Very Low Very Low
chemokine 6 5.00 Very Low Very Low Very Low
alcohol 5 5.00 Very Low Very Low Very Low
Multiple sclerosis 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Syndrome 38 100.00 Very High Very High Very High
Broca Aphasia 20 100.00 Very High Very High Very High
Cv General 4 Under Development 2 99.76 Very High Very High Very High
Apoptosis 55 98.14 Very High Very High Very High
Injury 47 95.98 Very High Very High Very High
Fatty Liver 24 85.72 High High
Liver Disease 43 82.84 Quite High
Stress 108 81.44 Quite High
Death 55 81.12 Quite High
Conversion Disorder 9 80.60 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We therefore conclude that in this patient, as in few other reported cases, the FAS is associated with agrammatism as a direct consequence of a cerebral infarct.


Positive_regulation (consequence) of FAS Binding (associated) of associated with cv general 4 under development, syndrome and broca aphasia
1) Confidence 0.50 Published 2007 Journal Ann Gen Psychiatry Section Body Doc Link PMC1774568 Disease Relevance 1.05 Pain Relevance 0.16
Overall, the most relevant mechanisms leading to hepatocyte injury in these metabolically altered conditions involve an excess of FFAs: directly inducing hepatocyte apoptosis and stimulating production of TNF, which is considered in this context as an adipocytokine; increasing Fas ligand binding to Fas (CD-95) receptor in steatotic hepatocytes, leading to apoptosis; leading to impaired mitochondrial or peroxisomal ?
Positive_regulation (increasing) of Fas Binding (binding) of in hepatocytes associated with injury and apoptosis
2) Confidence 0.11 Published 2008 Journal Fibrogenesis Tissue Repair Section Body Doc Link PMC2584013 Disease Relevance 1.58 Pain Relevance 0

General Comments

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