INT201871

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.01
First Reported 2006
Last Reported 2006
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.06
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
glands 1
PHOSPHO2 (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 28 5.00 Very Low Very Low Very Low
agonist 16 5.00 Very Low Very Low Very Low
endometriosis 3 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hyperplasia 4 57.84 Quite High
Uterine Fibroids 21 5.00 Very Low Very Low Very Low
Reprotox - General 1 11 5.00 Very Low Very Low Very Low
Endometriosis (extended) 9 5.00 Very Low Very Low Very Low
Hemorrhage 8 5.00 Very Low Very Low Very Low
Apoptosis 4 5.00 Very Low Very Low Very Low
Uterine Hemorrhage 2 5.00 Very Low Very Low Very Low
Gynecological Disease 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Congenital Anomalies 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, all doses of asoprisnil significantly suppressed the proliferation markers Ki-67 (Figure 5) and Phospho-H3 (Figure 6) in the endometrial glands, and the two higher doses caused significant shrinkage in endometrial thickness (Figures 7, 8) without inducing progestational effects such as glandular sacculation and secretion.
Negative_regulation (suppressed) of Phospho-H3 in glands
1) Confidence 0.01 Published 2006 Journal Reprod Biol Endocrinol Section Body Doc Link PMC1775068 Disease Relevance 0.06 Pain Relevance 0

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox