INT201944

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Context Info
Confidence 0.09
First Reported 2006
Last Reported 2006
Negated 1
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.79
Pain Relevance 0.55

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (CXCL10, AKT1) cell differentiation (AKT1) nucleoplasm (AKT1)
transport (AKT1) protein modification process (AKT1) extracellular space (CXCL10)
CXCL10 (Homo sapiens)
AKT1 (Homo sapiens)
Pain Link Frequency Relevance Heat
chemokine 29 99.68 Very High Very High Very High
cytokine 26 76.36 Quite High
Inflammation 14 75.96 Quite High
Arthritis 44 68.56 Quite High
spinal inflammation 19 59.36 Quite High
rheumatoid arthritis 22 55.52 Quite High
Inflammatory mediators 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
psoriasis 1 5.00 Very Low Very Low Very Low
Osteoarthritis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Necrosis 3 78.04 Quite High
Cancer 3 77.68 Quite High
INFLAMMATION 15 75.96 Quite High
Arthritis 27 68.56 Quite High
Arthropathy 3 65.96 Quite High
Low Back Pain 19 59.36 Quite High
Seronegative Spondarthritis 17 57.44 Quite High
Rheumatoid Arthritis 22 55.52 Quite High
Starvation 2 5.00 Very Low Very Low Very Low
Rheumatic Diseases 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In contrast to intact CXCL10, NH2-terminally truncated CXCL10(3–77) did not induce extracellular signal-regulated kinase 1/2 or Akt/protein kinase B phosphorylation in CXC chemokine receptor 3-transfected cells.
CXCL10 Neg (not) Positive_regulation (induce) of Phosphorylation (phosphorylation) of Akt associated with chemokine
1) Confidence 0.09 Published 2006 Journal Arthritis Res Ther Section Abstract Doc Link PMC1779382 Disease Relevance 0.79 Pain Relevance 0.55

General Comments

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