INT203766

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.00
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.99
Pain Relevance 0.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell proliferation (RETNLB) extracellular region (RETNLB) molecular_function (RETNLB)
cellular_component (RETNLB)
RETNLB (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 31 100.00 Very High Very High Very High
ischemia 1 36.56 Quite Low
depression 1 28.64 Quite Low
Inflammatory response 3 9.20 Low Low
cytokine 5 7.04 Low Low
Inflammatory mediators 1 6.60 Low Low
aspirin 2 5.00 Very Low Very Low Very Low
Angina 2 5.00 Very Low Very Low Very Low
Inflammatory marker 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 36 100.00 Very High Very High Very High
Infection 1 98.80 Very High Very High Very High
Pulmonary Disease 13 96.24 Very High Very High Very High
Pneumonia 2 92.08 High High
Bronchitis 1 91.48 High High
Nicotine Addiction 3 76.00 Quite High
Asthma 1 74.96 Quite High
Thrombosis 16 72.12 Quite High
Disease 4 38.48 Quite Low
Cv Unclassified Under Development 1 36.56 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The mechanism behind the augmentation of PM effect is unclear, but may be explained by changes in transcription of pro-inflammatory genes secondary to the acute infection.
Regulation (changes) of Transcription (transcription) of pro-inflammatory associated with inflammation and infection
1) Confidence 0.00 Published 2007 Journal Environ Health Section Body Doc Link PMC1800891 Disease Relevance 0.99 Pain Relevance 0.19

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox