INT205276

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Context Info
Confidence 0.01
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.66
Pain Relevance 0.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoskeletal protein binding (Plec) plasma membrane (Plec) cytoskeleton (Plec)
cytoplasm (Plec)
Plec (Mus musculus)
Pain Term Frequency Confidence Heat
Dynorphin 110 95.52 Very High Very High Very High
agonist 16 35.28 Quite Low
opioid receptor 10 34.92 Quite Low
Inflammation 4 10.64 Low Low
Endogenous opioid 5 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
adenocard 1 5.00 Very Low Very Low Very Low
ischemia 6 5.00 Very Low Very Low Very Low
Neurotransmitter 1 5.00 Very Low Very Low Very Low
Enkephalin 1 5.00 Very Low Very Low Very Low
Disease Term Frequency Confidence Heat
Stress 23 86.64 High High
Infection 9 81.76 Quite High
Sprains And Strains 24 73.68 Quite High
Critical Illness 3 66.28 Quite High
Disease 1 18.12 Low Low
Pulmonary Disease 1 14.88 Low Low
Burns 1 11.32 Low Low
INFLAMMATION 4 10.64 Low Low
Overdose 1 5.00 Very Low Very Low Very Low
Dislocations 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In the present work we show (Figure 8) that P. aeruginosa presents a “triple threat” to its host when exposed to dynorphin in that it can 1) activate QS circuits via enhanced PQS production, 2) reduce populations of protective probiotic bacteria such as lactobacilli through the production of HQNO and PCN [55], and 3) increase the production of QS-dependent virulence gene products that affect host cell function such as PCN and the PA-I lectin/adhesin [30,56–58].
QS-dependent virulence gene products Regulation (affect) of PCN associated with dynorphin
1) Confidence 0.01 Published 2007 Journal PLoS Pathogens Section Body Doc Link PMC1828698 Disease Relevance 0.66 Pain Relevance 0.11

General Comments

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