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Context Info
Confidence 0.43
First Reported 2007
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 5
Total Number 5
Disease Relevance 3.71
Pain Relevance 1.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (LPA) transport (LPA) small molecule metabolic process (LPA)
extracellular region (LPA) lipid metabolic process (LPA)
LPA (Homo sapiens)
Pain Link Frequency Relevance Heat
Cancer pain 20 99.92 Very High Very High Very High
nociceptor 6 95.24 Very High Very High Very High
Kinase C 37 80.60 Quite High
dorsal root ganglion 53 79.52 Quite High
Inflammation 59 76.56 Quite High
cytokine 28 71.32 Quite High
Neuropathic pain 10 61.56 Quite High
Inflammatory response 8 57.96 Quite High
nav1.8 2 38.36 Quite Low
Pain 12 36.64 Quite Low
Disease Link Frequency Relevance Heat
Disorder Of Lipid Metabolism 33 100.00 Very High Very High Very High
Repression 2 100.00 Very High Very High Very High
Cancer Pain 3 99.92 Very High Very High Very High
Cancer 54 96.28 Very High Very High Very High
Bone Cancer 43 94.00 High High
Hyperlipidemia 4 87.88 High High
Insulin Resistance 10 85.76 High High
Obesity 10 85.76 High High
Diabetes Mellitus 86 84.00 Quite High
Myocardial Infarction 29 83.48 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
To test for differences between Lp(a) tertiles, categorical variables were compared using the ?
Negative_regulation (differences) of Gene_expression (tertiles) of Lp
1) Confidence 0.43 Published 2010 Journal Korean Circulation Journal Section Body Doc Link PMC2978291 Disease Relevance 0.71 Pain Relevance 0
Similarly, LPA stimulated production of inositol phosphates reached a maximal level at 1 ?
Negative_regulation (reached) of Gene_expression (production) of LPA
2) Confidence 0.22 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2621239 Disease Relevance 0 Pain Relevance 0
Thus, the lower levels of Lp(a) in patients with elevated triglyceride could be the result of the rapid catabolism of TRP apo(a) compared with the slower apo(a) catabolism in the LDL density range [17].
Negative_regulation (catabolism) of Gene_expression (result) of Lp
3) Confidence 0.07 Published 2007 Journal Lipids Health Dis Section Body Doc Link PMC2216007 Disease Relevance 0.78 Pain Relevance 0
Inhibition of the LPA1 receptor or synthesis of LPA may be a novel therapy for cancer pain.

Negative_regulation (Inhibition) of Gene_expression (synthesis) of LPA associated with cancer pain
4) Confidence 0.02 Published 2010 Journal Mol Pain Section Body Doc Link PMC3004845 Disease Relevance 1.22 Pain Relevance 0.72
The effects that PPAR alpha exerts on triglyceride-rich lipoproteins is due to their stimulation of lipoprotein lipase and repression of apolipoprotein CIII expression, while the effects on high-density lipoproteins depend upon the regulation of apolipoproteins AI and AII.
Negative_regulation (repression) of Gene_expression (expression) of apolipoprotein CIII associated with repression and disorder of lipid metabolism
5) Confidence 0.00 Published 2007 Journal Immun Ageing Section Body Doc Link PMC1845171 Disease Relevance 1.00 Pain Relevance 0.31

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