INT206193

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Context Info
Confidence 0.35
First Reported 2007
Last Reported 2011
Negated 0
Speculated 0
Reported most in Body
Documents 6
Total Number 6
Disease Relevance 2.24
Pain Relevance 4.06

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Ltp (Mus musculus)
Pain Link Frequency Relevance Heat
long-term potentiation 295 100.00 Very High Very High Very High
amygdala 198 100.00 Very High Very High Very High
qutenza 79 100.00 Very High Very High Very High
depression 33 100.00 Very High Very High Very High
noradrenaline 8 98.68 Very High Very High Very High
gABA 10 95.48 Very High Very High Very High
Glutamate 18 95.16 Very High Very High Very High
adenocard 5 89.52 High High
Dopamine 8 85.24 High High
Hippocampus 44 81.60 Quite High
Disease Link Frequency Relevance Heat
Depression 33 100.00 Very High Very High Very High
Anxiety Disorder 74 97.72 Very High Very High Very High
Stress 43 96.96 Very High Very High Very High
Cognitive Disorder 116 88.80 High High
Alzheimer's Dementia 15 64.80 Quite High
Targeted Disruption 43 53.48 Quite High
Neuroblastoma 1 45.52 Quite Low
Disease 39 5.00 Very Low Very Low Very Low
Amyloid Plaque 14 5.00 Very Low Very Low Very Low
Pain 7 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
NO and CB1 receptors are involved in mediating capsaicin-induced reduction of LA-LTP
Regulation (involved) of Localization (reduction) of LTP associated with qutenza, long-term potentiation and amygdala
1) Confidence 0.35 Published 2011 Journal PLoS ONE Section Body Doc Link PMC3020947 Disease Relevance 0 Pain Relevance 1.43
releasing hormone [34] increase LTP, the abundance of these hormones relative to that of corticosterone is very important in
Regulation (important) of Localization (releasing) of LTP associated with long-term potentiation
2) Confidence 0.23 Published 2007 Journal Neural Plasticity Section Body Doc Link PMC1847472 Disease Relevance 0.60 Pain Relevance 0.74
A persistent late phase of LTP (L-LTP) in cortical and thalamic inputs to the LA, which is induced by repeated high frequency trains of presynaptic stimulation and is dependent on new protein synthesis, was found to be mediated by activation of ?
Regulation (dependent) of Localization (phase) of LTP associated with long-term potentiation
3) Confidence 0.22 Published 2010 Journal Mol Brain Section Body Doc Link PMC2877027 Disease Relevance 0.41 Pain Relevance 0.55
A persistent late phase of LTP (L-LTP) in cortical and thalamic inputs to the LA, which is induced by repeated high frequency trains of presynaptic stimulation and is dependent on new protein synthesis, was found to be mediated by activation of ?
Regulation (dependent) of Localization (phase) of L-LTP associated with long-term potentiation
4) Confidence 0.22 Published 2010 Journal Mol Brain Section Body Doc Link PMC2877027 Disease Relevance 0.41 Pain Relevance 0.55
Accordingly, it is now evident that, besides excitatory strengthening mechanisms in LTP, stabilization or suppression mechanisms, e.g., LTD, are also crucial for the regulation of synaptic plasticity (Abel et al. 1998).
Regulation (crucial) of Localization (stabilization) of LTP associated with depression and long-term potentiation
5) Confidence 0.18 Published 2008 Journal Psychopharmacology (Berl) Section Body Doc Link PMC2704616 Disease Relevance 0.60 Pain Relevance 0.55
oligomers and mediating downstream synaptic response is not clear, as conflicting results have been reported on LTP in A?
Regulation (on) of Localization (reported) of LTP associated with long-term potentiation
6) Confidence 0.18 Published 2010 Journal Neuropsychiatric Disease and Treatment Section Body Doc Link PMC2938309 Disease Relevance 0.23 Pain Relevance 0.24

General Comments

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