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Context Info
Confidence 0.11
First Reported 2007
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 0.63
Pain Relevance 0.49

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell death (Nlrp1a) nucleus (Nlrp1a) enzyme binding (Nlrp1a)
Nlrp1a (Mus musculus)
Pain Link Frequency Relevance Heat
Nucleus accumbens 24 100.00 Very High Very High Very High
Dopamine 17 77.60 Quite High
Morphine 49 71.24 Quite High
Ventral tegmentum 5 64.20 Quite High
Catechol-O-methyltransferase 46 46.76 Quite Low
amygdala 3 41.68 Quite Low
Hippocampus 1 18.56 Low Low
Substantia nigra 20 5.00 Very Low Very Low Very Low
cocaine 9 5.00 Very Low Very Low Very Low
long-term potentiation 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Sprains And Strains 83 95.24 Very High Very High Very High
Targeted Disruption 26 86.88 High High
Disease 28 63.56 Quite High
Urological Neuroanatomy 10 62.72 Quite High
Toxicity 8 55.76 Quite High
Parkinson's Disease 4 55.40 Quite High
Stress 12 8.32 Low Low
Depression 5 5.00 Very Low Very Low Very Low
Drug Dependence 3 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This loss of TH+ terminals was significantly attenuated by chronic oral NAC supplementation beginning at 3 weeks of age (p?
Transcription (beginning) of NAC
1) Confidence 0.11 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2925900 Disease Relevance 0.32 Pain Relevance 0.04
Of importance to the current study is that Kerns et al. have recently compared mRNA expression in the NAC in control C57 and DBA animals [16].
Transcription (expression) of NAC associated with nucleus accumbens
2) Confidence 0.09 Published 2007 Journal BMC Genomics Section Body Doc Link PMC1851712 Disease Relevance 0.31 Pain Relevance 0.46

General Comments

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