INT207846

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Context Info
Confidence 0.06
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.14
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (INS) small molecule metabolic process (INS) extracellular region (INS)
carbohydrate metabolic process (INS) cell-cell signaling (INS)
Anatomy Link Frequency
brain 1
Hypism (Mus musculus)
INS (Homo sapiens)
Pain Link Frequency Relevance Heat
Hippocampus 2 85.00 Quite High
Inflammation 10 5.00 Very Low Very Low Very Low
tolerance 2 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
cINOD 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Intracerebroventricular 1 5.00 Very Low Very Low Very Low
addiction 1 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hyperinsulinism 14 100.00 Very High Very High Very High
Insulin Resistance 103 98.20 Very High Very High Very High
Hypoglycemia 3 95.64 Very High Very High Very High
Diabetes Mellitus 24 68.56 Quite High
Birth Weight 11 41.12 Quite Low
Disease 8 15.68 Low Low
Metabolic Syndrome 46 5.00 Very Low Very Low Very Low
Obesity 15 5.00 Very Low Very Low Very Low
Aging 12 5.00 Very Low Very Low Very Low
INFLAMMATION 12 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Hyperinsulinemia is known to down-regulate insulin receptor activity in peripheral tissues but not in the nervous tissue [77] and this differential loss of sensitivity presumably enhances brain activity at the cost of muscle activity.
Hyperinsulinemia Regulation (regulate) of insulin in brain associated with hyperinsulinism
1) Confidence 0.06 Published 2007 Journal BMC Evol Biol Section Body Doc Link PMC1868084 Disease Relevance 1.14 Pain Relevance 0.04

General Comments

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