INT208526

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Context Info
Confidence 0.18
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.25
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (SCGB1A1) extracellular region (SCGB1A1) cytoplasm (SCGB1A1)
Anatomy Link Frequency
neutrophil 3
monocyte 1
SCGB1A1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 2 5.00 Very Low Very Low Very Low
Dismenorea 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Infection 4 64.00 Quite High
Bacterial Respiratory Disease 2 60.72 Quite High
Stress 2 41.76 Quite Low
Reprotox - General 1 4 5.00 Very Low Very Low Very Low
Dysmenorrhea 2 5.00 Very Low Very Low Very Low
Hyperplasia 2 5.00 Very Low Very Low Very Low
Hypertrophy 2 5.00 Very Low Very Low Very Low
Flushing 2 5.00 Very Low Very Low Very Low
Leukemia 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
An immunomodulatory role has also been suggested for uteroglobin, an unusually small globular protein that is produced in maximal quantities during the secretory phase and is a potent inhibitor of neutrophil and monocyte chemotaxis in vitro [34,35].
Negative_regulation (inhibitor) of Gene_expression (produced) of uteroglobin in neutrophil
1) Confidence 0.18 Published 2007 Journal Placenta Section Body Doc Link PMC1878510 Disease Relevance 0.12 Pain Relevance 0
An immunomodulatory role has also been suggested for uteroglobin, an unusually small globular protein that is produced in maximal quantities during the secretory phase and is a potent inhibitor of neutrophil and monocyte chemotaxis in vitro [34,35].
Negative_regulation (inhibitor) of in monocyte Gene_expression (produced) of uteroglobin in neutrophil
2) Confidence 0.06 Published 2007 Journal Placenta Section Body Doc Link PMC1878510 Disease Relevance 0.12 Pain Relevance 0

General Comments

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