INT209195

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Context Info
Confidence 0.26
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

aging (IL1B) transmembrane transport (TRPC3) cell-cell signaling (IL1B)
cytosol (IL1B) extracellular space (IL1B) signal transduction (IL1B)
IL1B (Homo sapiens)
TRPC3 (Homo sapiens)
Pain Link Frequency Relevance Heat
Calcium channel 1 99.48 Very High Very High Very High
cytokine 6 74.04 Quite High
agonist 1 56.24 Quite High
member 8 1 37.00 Quite Low
Inflammation 7 26.88 Quite Low
Inflammatory mediators 2 15.48 Low Low
metalloproteinase 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pre-term Labor 1 36.80 Quite Low
INFLAMMATION 10 26.88 Quite Low
Infection 3 5.00 Very Low Very Low Very Low
Syndrome 2 5.00 Very Low Very Low Very Low
Necrosis 1 5.00 Very Low Very Low Very Low
Cancer 1 5.00 Very Low Very Low Very Low
Bacterial Vaginosis 1 5.00 Very Low Very Low Very Low
Disease Progression 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Specifically, IL1B modulates protein expression of TRPC3 (a putative calcium channel) and SR Ca2+-ATPases, initiates spontaneous [Ca2+]i oscillations and augments calcium influx pathways and a store operated current [26,28].
IL1B Regulation (modulates) of Gene_expression (expression) of TRPC3 associated with calcium channel
1) Confidence 0.26 Published 2007 Journal BMC Pregnancy Childbirth Section Body Doc Link PMC1892065 Disease Relevance 0 Pain Relevance 0.11

General Comments

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