INT209196

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Context Info
Confidence 0.68
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.23
Pain Relevance 0.15

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

aging (IL1B) transmembrane transport (TRPC3) cell-cell signaling (IL1B)
cytosol (IL1B) extracellular space (IL1B) signal transduction (IL1B)
Anatomy Link Frequency
uterine muscle 2
IL1B (Homo sapiens)
TRPC3 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 6 94.36 High High
Calcium channel 1 77.88 Quite High
Inflammation 7 76.96 Quite High
Inflammatory mediators 2 45.20 Quite Low
metalloproteinase 1 38.96 Quite Low
agonist 1 31.32 Quite Low
Inflammatory response 1 29.60 Quite Low
member 8 1 12.08 Low Low
Disease Link Frequency Relevance Heat
Pre-term Labor 1 93.44 High High
INFLAMMATION 10 76.96 Quite High
Cancer 1 41.76 Quite Low
Necrosis 1 41.20 Quite Low
Infection 3 5.00 Very Low Very Low Very Low
Syndrome 2 5.00 Very Low Very Low Very Low
Bacterial Vaginosis 1 5.00 Very Low Very Low Very Low
Disease Progression 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The induction of TRPC3 protein expression by IL1B may in part explain the increase in TRPC3 expression observed in uterine muscle at labour onset and provides a plausible mechanism by which cytokines could induce premature labour.
IL1B Positive_regulation (induction) of Gene_expression (expression) of TRPC3 in uterine muscle associated with pre-term labor and cytokine
1) Confidence 0.68 Published 2007 Journal BMC Pregnancy Childbirth Section Body Doc Link PMC1892065 Disease Relevance 0.23 Pain Relevance 0.15

General Comments

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