INT209390

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Context Info
Confidence 0.20
First Reported 2007
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 4
Total Number 5
Disease Relevance 3.97
Pain Relevance 1.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transferase activity, transferring glycosyl groups (Ugcg) Golgi apparatus (Ugcg) lipid metabolic process (Ugcg)
Anatomy Link Frequency
brain 4
neurons 2
decidua 2
Ugcg (Mus musculus)
Pain Link Frequency Relevance Heat
medulla 53 100.00 Very High Very High Very High
central sensitization 1 97.76 Very High Very High Very High
cva 16 86.72 High High
Dismenorea 1 78.00 Quite High
Dopamine 3 17.80 Low Low
monoamine 13 16.16 Low Low
Neuropeptide 1 6.80 Low Low
Central nervous system 19 5.00 Very Low Very Low Very Low
Pain 17 5.00 Very Low Very Low Very Low
Catecholamine 8 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 135 100.00 Very High Very High Very High
Brain Death 94 100.00 Very High Very High Very High
Infection 50 100.00 Very High Very High Very High
Cough 10 99.36 Very High Very High Very High
Coma 24 98.72 Very High Very High Very High
Body Weight 7 98.48 Very High Very High Very High
Sleep Disorders 6 89.00 High High
Syndrome 1 87.84 High High
Hypothermia 4 87.52 High High
Brain Injury 10 87.12 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thus, we hypothesize that shipment also exaggerates the HPA axis responsiveness for approximately three weeks due to central sensitization of CRF-releasing neurons in the pPVN followed by a phase of reduced CRF release four weeks after shipment that prevents an acute stress-induced release of GCs.


Negative_regulation (prevents) of Localization (release) of GCs in neurons associated with stress and central sensitization
1) Confidence 0.20 Published 2010 Journal BMC Physiol Section Body Doc Link PMC2845127 Disease Relevance 0.88 Pain Relevance 0.05
GCs in the tegument of T. solium metacestodes are also released during infection
Negative_regulation (infection) of Localization (released) of GCs associated with infection
2) Confidence 0.06 Published 2008 Journal PLoS Neglected Tropical Diseases Section Body Doc Link PMC2274955 Disease Relevance 0.52 Pain Relevance 0
It is also notable that the GCs migrating into the DB are strongly immunoreactive for COX, and so PGs synthesised by these cells could exert local paracrine effects in the decidua.
Negative_regulation (immunoreactive) of Localization (migrating) of GCs in decidua
3) Confidence 0.06 Published 2007 Journal Placenta Section Body Doc Link PMC1895600 Disease Relevance 0.49 Pain Relevance 0.08
First, based on the GCS and examination of brainstem reflexes, imminent brain death can be defined as: ‘A state in which a deeply comatose, mechanically ventilated patient, admitted to an ICU, with irreversible catastrophic brain damage of known origin (e.g.
Negative_regulation (defined) of Localization (based) of GCS in brain associated with brain death, medulla and coma
4) Confidence 0.03 Published 2010 Journal Intensive Care Med Section Body Doc Link PMC2921050 Disease Relevance 1.04 Pain Relevance 0.49
First, based on the GCS and examination of brainstem reflexes, imminent brain death can be defined as: ‘A state in which a deeply comatose, mechanically ventilated patient, admitted to an ICU, with irreversible catastrophic brain damage of known origin (e.g.
Negative_regulation (defined) of Localization (examination) of GCS in brain associated with brain death, medulla and coma
5) Confidence 0.03 Published 2010 Journal Intensive Care Med Section Body Doc Link PMC2921050 Disease Relevance 1.03 Pain Relevance 0.49

General Comments

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