INT210551

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Context Info
Confidence 0.08
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (KCNH2, KCNE2) nuclear envelope (KCNH2) lysosome (KCNE2)
transmembrane transport (KCNH2) cytoplasm (KCNH2)
Anatomy Link Frequency
cardiac conduction system 1
KCNH2 (Homo sapiens)
KCNE2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Action potential 8 92.72 High High
Potency 8 76.48 Quite High
tricyclic antidepressant 7 50.00 Quite Low
potassium channel 1 50.00 Quite Low
addiction 23 5.00 Very Low Very Low Very Low
Endep 7 5.00 Very Low Very Low Very Low
sSRI 2 5.00 Very Low Very Low Very Low
antidepressant 2 5.00 Very Low Very Low Very Low
antiarrhythmic agent 1 5.00 Very Low Very Low Very Low
methadone 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 1 13.40 Low Low
Arrhythmia Under Development 4 5.00 Very Low Very Low Very Low
Overdose 3 5.00 Very Low Very Low Very Low
Syndrome 3 5.00 Very Low Very Low Very Low
Syncope 1 5.00 Very Low Very Low Very Low
Congenital Anomalies 1 5.00 Very Low Very Low Very Low
Sudden Death 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
subunit suggested to be necessary to recapitulate native IKr [55], it has recently been suggested that MiRP1 is unlikely to interact with HERG outside of the cardiac conduction system [51] and, additionally, the pharmacological sensitivity of HERG channels expressed in mammalian cells without MiRP1 co-expression has been found to be similar to that of native IKr [56].
HERG Binding (interact) of MiRP1 in cardiac conduction system
1) Confidence 0.08 Published 2007 Journal Biochem Pharmacol Section Body Doc Link PMC1920586 Disease Relevance 0 Pain Relevance 0.19

General Comments

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