INT211291

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Context Info
Confidence 0.12
First Reported 2007
Last Reported 2010
Negated 1
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 0.97
Pain Relevance 0.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
NK cells 2
T cells 2
Trav6-3 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 28 98.68 Very High Very High Very High
rheumatoid arthritis 26 77.04 Quite High
cytokine 55 5.00 Very Low Very Low Very Low
chemokine 8 5.00 Very Low Very Low Very Low
Arthritis 8 5.00 Very Low Very Low Very Low
Inflammatory response 4 5.00 Very Low Very Low Very Low
methotrexate 2 5.00 Very Low Very Low Very Low
withdrawal 2 5.00 Very Low Very Low Very Low
cINOD 2 5.00 Very Low Very Low Very Low
Rheumatism 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 32 98.68 Very High Very High Very High
Disease 28 92.40 High High
Hematologic Neoplasms 2 80.32 Quite High
Solid Tumor 2 79.16 Quite High
Systemic Lupus Erythematosus 2 78.36 Quite High
Rheumatoid Arthritis 26 77.04 Quite High
Autoimmune Disease 4 75.88 Quite High
Mycobacterial Infection 2 75.04 Quite High
Acquired Immune Deficiency Syndrome Or Hiv Infection 2 73.84 Quite High
Infection 27 69.96 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
is rate-limiting, thereby regulating the amount of TCR/CD3 complex expressed at the cell surface.8,9 In NK cells, the TCR?
Regulation (amount) of TCR Binding (complex) of in NK cells
1) Confidence 0.12 Published 2007 Journal Blood Section Body Doc Link PMC1939810 Disease Relevance 0 Pain Relevance 0
may serve to attenuate T-cell activation at sites of tissue damage, thereby limiting the effects of unbridled T-cell reactivity and pathologic effector responses.14,15 Whilst this model is entirely consistent with a central role for antigen in driving autoimmune inflammatory activity, it implies that chronically activated T cells rendered hyporesponsive to TCR engagement through loss of TCR?
Regulation (hyporesponsive) of TCR Binding (engagement) of in T cells associated with inflammation
2) Confidence 0.12 Published 2007 Journal Blood Section Body Doc Link PMC1939810 Disease Relevance 0.84 Pain Relevance 0.11
Interaction between the nine NP366 peptide variants and the public TCR did not result in the production of IL-2 under the same experimental conditions.
Neg (not) Regulation (result) of TCR Binding (Interaction) of
3) Confidence 0.07 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2868023 Disease Relevance 0.14 Pain Relevance 0

General Comments

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