INT212789

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Context Info
Confidence 0.38
First Reported 2007
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.04
Pain Relevance 0.17

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (C5) extracellular region (C5) response to stress (C5)
Anatomy Link Frequency
neutrophil 2
C5 (Homo sapiens)
Pain Link Frequency Relevance Heat
rheumatoid arthritis 76 97.64 Very High Very High Very High
Arthritis 15 5.00 Very Low Very Low Very Low
Inflammation 14 5.00 Very Low Very Low Very Low
cINOD 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
Analgesic 1 5.00 Very Low Very Low Very Low
Rheumatism 1 5.00 Very Low Very Low Very Low
Inflammatory response 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 37 97.96 Very High Very High Very High
Rheumatoid Arthritis 77 97.64 Very High Very High Very High
Sprains And Strains 1 88.32 High High
Infection 8 85.12 High High
Pneumonia 1 31.84 Quite Low
Targeted Disruption 2 28.32 Quite Low
Disease Progression 2 24.60 Low Low
INFLAMMATION 16 5.00 Very Low Very Low Very Low
Arthritis 14 5.00 Very Low Very Low Very Low
Hemolysis 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Our study defines the TRAF1/C5 as a novel genetic region present in the human genome that predisposes to RA.
Negative_regulation (defines) of Gene_expression (/) of C5 associated with rheumatoid arthritis
1) Confidence 0.38 Published 2007 Journal PLoS Medicine Section Body Doc Link PMC1976626 Disease Relevance 0.87 Pain Relevance 0.17
Modulation of the complement effector functions: inhibiting C5 activation and neutrophil migration
Negative_regulation (inhibiting) of Gene_expression (activation) of C5 in neutrophil
2) Confidence 0.12 Published 2010 Journal J Mol Med Section Body Doc Link PMC2832872 Disease Relevance 0.17 Pain Relevance 0

General Comments

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