INT213043

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Context Info
Confidence 0.05
First Reported 2005
Last Reported 2005
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.12
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

lipid binding (Apoa1) extracellular space (Apoa1) extracellular region (Apoa1)
cell adhesion (Scarb1) plasma membrane (Scarb1) nucleus (Apoa1)
Anatomy Link Frequency
endothelial cells 2
Apoa1 (Mus musculus)
Scarb1 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 11 30.48 Quite Low
metalloproteinase 6 5.00 Very Low Very Low Very Low
cytokine 4 5.00 Very Low Very Low Very Low
Tetrahydrobiopterin 3 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
chemokine 2 5.00 Very Low Very Low Very Low
COX2 1 5.00 Very Low Very Low Very Low
Angina 1 5.00 Very Low Very Low Very Low
Bioavailability 1 5.00 Very Low Very Low Very Low
Bile 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disorder Of Lipid Metabolism 137 99.32 Very High Very High Very High
Necrosis 3 61.64 Quite High
Cancer 1 58.24 Quite High
Apoptosis 7 57.56 Quite High
INFLAMMATION 19 50.00 Quite Low
Death 5 47.60 Quite Low
Atherosclerotic Plaque 3 18.08 Low Low
Adhesions 18 5.00 Very Low Very Low Very Low
Atherosclerosis 10 5.00 Very Low Very Low Very Low
Coronary Artery Disease 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Incubation of cultured endothelial cells with HDL activates eNOS in a process that involves the binding of ApoA-I to the scavenger receptor-BI (SR-BI) (Yuhanna et al 2001; Mineo et al 2003).
Regulation (involves) of ApoA-I Binding (binding) of scavenger receptor-BI in endothelial cells associated with disorder of lipid metabolism
1) Confidence 0.05 Published 2005 Journal Vascular Health and Risk Management Section Body Doc Link PMC1993938 Disease Relevance 1.12 Pain Relevance 0

General Comments

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