INT213786

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Context Info
Confidence 0.27
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.92
Pain Relevance 0.46

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (F2rl3) plasma membrane (F2rl3) signal transducer activity (F2rl3)
Anatomy Link Frequency
bladder 2
F2rl3 (Mus musculus)
Pain Link Frequency Relevance Heat
substance P 3 99.50 Very High Very High Very High
Inflammatory stimuli 3 98.98 Very High Very High Very High
Inflammatory response 7 97.94 Very High Very High Very High
Inflammation 43 92.16 High High
Pain 2 61.48 Quite High
antagonist 1 35.28 Quite Low
interstitial cystitis 4 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
Inflammatory mediators 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 54 98.82 Very High Very High Very High
Overactive Bladder 11 86.44 High High
Pain 2 61.48 Quite High
Injury 3 59.28 Quite High
Cystitis 8 51.68 Quite High
Bladder Cancer 2 51.16 Quite High
Cancer 1 43.36 Quite Low
Urinary Dysfunction 1 38.56 Quite Low
Disease 2 26.08 Quite Low
Microphthalmia 23 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Additional evidence for the participation of PARs in the bladder inflammatory response was the finding that known pro-inflammatory stimuli, such as LPS, substance P, and antigen challenge, induce an increase in PAR4 RNA within hours of stimulation [23].
Positive_regulation (induce) of Positive_regulation (increase) of PAR4 in bladder associated with inflammatory response, substance p and inflammatory stimuli
1) Confidence 0.27 Published 2007 Journal BMC Immunol Section Body Doc Link PMC2000913 Disease Relevance 0.92 Pain Relevance 0.46

General Comments

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